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Additional info for Clinical Exercise and Testing (European Respiratory Monograph)
Oxidative energy supply. In: Energy Metabolism of Human Muscle. Baltimore, University Park Press, 1972; pp. 52–202. Brown GC. Control of respiration and ATP synthesis in mammalian mitochondria and cells. Biochem J 1992; 284: 1–13. Meyer RA, Foley JM. Cellular processes integrating the metabolic response to exercise. In: Rowell LB, Shepherd Jt, eds. Handbook of Physiology. Section 12. Exercise: Regulation and Integration of Multiple Systems. Bethesda, Amer Physiol Soc, 1996; pp. 841–869. Kushmerick MJ, Conley KE.
In the simplest form, this means that the instantaneous rate of change of the particular variable will be proportional to the distance from the steady state. The time to attain a steady state naturally depends upon the rate at which this exponential response develops. This is conventionally characterised with respect to the time constant (t) of the response. For such an exponential response, this is the time for the variable to attain 63% of the final steady-state change. e. the function will have attained 99% of its steady-state value, which is well within current levels of discriminability.
Dynamics of breathing during exercise. In: Whipp BJ, Wasserman K. Pulmonary Physiology and Pathophysiology of Exercise. New York, Marcel Decker Ltd, 1991; pp. 67–97. Ward SA, Whipp BJ. Influence of body CO2 stores on ventilatory-metabolic coupling during exercise. In: Honda Y, Miyamoto Y, Konno K, Widdicombe JG, eds. Control of Breathing and its Modeling Perspective. New York, Plenum Press, 1992; pp. 425–431. Rausch SM, Whipp BJ, Wasserman K, Huszezuk A. Role of the carotid bodies in the respiratory compensation for the metabolic acidosis of exercise in humans.