By Janet E. Hall, Lynnette K. Nieman
Skilled physicians concisely clarify the pathophysiology and scientific manifestations of endocrine issues and survey the entire most modern laboratory diagnostics. issues diversity largely from an summary of the prognosis of diabetes and the long term tracking of its problems to the review of menstrual disorder. insurance can also be given to the prognosis of pituitary tumors, Cushing's syndrome, thyroid ailment, and hypoglycemia; the evaluate of endocrine-induced high blood pressure; the review of dyslipidemia and weight problems; and methods to diagnosing hyper- and hypocalcemia. There also are discussions of osteoporosis, hypogonadism and erectile disorder, and hyperandrogenism in ladies. The authors every one assessment the advanced physiological foundation of the correct endocrine techniques and supply richly instructive concepts for followup and long term administration of sufferers.
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Extra info for Handbook of Diagnostic Endocrinology (Contemporary Endocrinology)
Spratt DI, O’Dea LS, Schoenfeld DA, Butler J, Rao PN, Crowley WF Jr. Neuroendocrinegonadol axis in men: frequent sampling of LH, FSH and testosterone. Am J Physiol 1988;254:E658–E666. 20. Santoro N, Butler JP, Filicori M, Crowley WF Jr. Alterations of the hypothalamic GnRH interpulse interval sequence over the normal menstrual cycle. Am J Physiol 1988;255:E696–701. 01/Sluss/1-22/F 20 12/2/02, 8:53 AM Chapter 1/Endocrine Immunoassay 21 21. Veldhuis JD, Beitins IZ, Johnson ML, Serabian MA, Dufau ML.
Severe nephrogenic DI is most commonly congenital, due to defects in the gene 02/Verbalis/23-54/F 38 12/2/02, 8:36 AM Chapter 2/Water Metabolism Disorders 39 Table 2 Common Etiologies of Polydipsia and Hypotonic Polyuria Central (neurogenic) diabetes insipidus Congenital (congenital malformations; autosomal dominant: AVP-neurophysin gene mutations). Drug/toxin-induced (ethanol, diphenylhydantoin, snake venom). Granulomatous (histiocytosis, sarcoidosis). Neoplastic (craniopharyngioma, meningioma, germinoma, pituitary tumor, or metastases).
Because of the importance of Na+ for ensuring maintenance of the ECF volume, which in turn directly supports blood volume and pressure, its uniqueness insofar as meriting a specific mechanism for regulated intake seems appropriate. However, despite abundant evidence in many different species demonstrating a salt appetite that is proportionately related to Na+ losses (19), there is only one pathological condition in which a specific stimulated sodium appetite has been unequivocally observed in humans, namely Addison’s disease, which is caused by adrenal insufficiency.