By S.K. Gill, M.M. Brown, F. Robertson, N. Losseff
This selection of case reviews is authored through a bunch of the world over acknowledged specialists within the box of cerebrovascular affliction. The circumstances are rigorously chosen to supply an perception into different forms of stroke syndromes from the typical via to extra infrequent displays of cerebrovascular pathology. each one case is labored via with a dialogue on presentation, scientific positive aspects, exam, radiological findings and administration. eventually, studying issues are highlighted to assist the reader specialize in the major issues of the case research.
Stroke medication: Case Studiesfrom Queen Square is geared toward clinical scholars and medical professionals in all levels of teaching from starting place years via to consultancy.
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Extra resources for Stroke Medicine: Case Studies from Queen Square
Lancet Neurol. 2011;10(3):264–74. 2. Guo DC, Papke CL, Tran-Fadulu V, et al. Mutations in smooth muscle alpha-actin (ACTA2) cause coronary artery disease, stroke, and moyamoya disease, along with thoracic aortic disease. Am J Hum Genet. 2009;84(5):617–27. 3 A Child with Enlarged Pupils 21 3. Milewicz DM, Østergaard JR, Ala-Kokko LM, et al. De novo ACTA2 mutation causes a novel syndrome of multisystemic smooth muscle dysfunction. Am J Med Genet A. 2010;152A(10):2437–43. 4. Munot P, Saunders DE, Milewicz DM, et al.
2009;8:724–30. 20. Mayer TE, Hamann GF, Brueckman HJ. Treatment of basilar artery embolism with a mechanical extraction device: necessity of flow reversal. Stroke. 2002;33:2232–5. 21. Zaidat OO, Tolbert M, Smith TP, et al. Primary endovascular therapy with clot retrieval and balloon angioplasty for acute basilar artery occlusion. Pediatr Neurosurg. 2005;41:323–7. 22. Yu W, Binder D, Foster-Barber A, et al. Endovascular embolectomy of acute basilar artery occlusion. Neurology. 2003;61:1421–3. 23.
1007/978-1-4471-6705-1_5 31 32 D. Doig and F. Kennedy Examination After recovery from the seizure, examination of the central nervous system was unremarkable apart from some generally brisk reflexes. Of note there was no limb weakness on examination of the peripheral nervous system, and no speech disturbance. The patient was orientated in time and date, but was not able to give the name or location of the hospital. Neuropsychological testing revealed marked cognitive decline for his age group relative to an estimated low average/average predicted pre-morbid estimated ability.